dr mohamed saker posts
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Talta Fopcu لو محدش جاوب هنخلي الدكتور محمد صقر لما يقعد يجاوب إن شاء الله
Mohamed Sakr
about an hour ago · ·
1
Mohamed Sakr alpha 1 agonist contra'indicated in cardiogenic shock due to present of decreasing blood flow to organs and alpha 1 agonist make vasoconstriction decreasing blood flow to organs .
27 minutes ago · · 1
Mohamed Sakr septic shock is presence of infection in the bovy making release of inflammatory madiators making severe vasodilation making hypotension must give alpha 1 agonist to make vasoconstriction and increasing blood pressure
25 minutes ago · · 1
Mohamed Sakr dopamine is first choice in treatment of cardiogenic shock due to making increasing cardiac output through acting on beta 1 receptor and making vasodilation of blood vessels making increase of blood flow through acting on dopamine receptor
21 minutes ago · · 1
Mohamed Sakr betablocker treatment hypertension due to blocking beta 1 receptor in the heart making decreasing in heart rate + decreasing cardiac output and blocking of bata 1 receptor in the kidney juxtaglomerular cells decreasing renin and decreasing angiotensin II make V.D + blocking of beta 2 receptor in the sympathetic nerve decreasing NA release + inhibit sympathetic outflow centrally from sympathetic center
17 minutes ago · · 1
Mohamed Sakr use carvidelol , metoprolol , bisoprolol in treatment of heart failure due to prevention of excessive sympathetic discharge on the heart + carvidelol making alpha blocker activity making V.D in artery and veins making decreasing afterload and preload making resting of heart
12 minutes ago · · 1- Talta Fopcu Mohamed Sakr
in phaechromocytoma tumour in adrenal medulla treatment by alpha blocker to decreasing B.P + use beta blocker to prevent tachycardia like labetalol .50 minutes ago · - Talta Fopcu Kholoud Osama
hwa eh el far2 ben el ( stable angina ) and ( varient angina ) ?
Mohamed Sakr
stable angina is effort angina cause due to atherosclerosis in coronary arteries making loss of elasticity in coronaries when exert effort increasing heart work with no increase in oxygen blodd supply from coronaries leadsto pain of angina to treatment it give beta blocker to block beta1 receptor in the heart to make decreasing heart work to prevent occuranceof angina
Mohamed Sakr
variant angina has not atherosclerosis making by vasoconstriction of lcoronary artery during rest must treatment by vasodilator like ca+2 channel blocker + nitrates and contra'indicated use of beta blocker due to block beta 2 receptor in the coronaries making predominates effect of alpha 1 receptor in the blood vessel making vasoconstriction of coronaries making increase in angina symptoms .49 minutes ago · 
Rania Mohammed myocardial infarction is death of part of the heart due to thrombus in coronary artery which supply heart by oxygenated blood while heart failure is decreasing contractility of the heart not able to pump the blood to the body :48 minutes ago · · 1- Talta Fopcu Rania Mohammed
هو ليه ال alpha 1 agonists بنستخدمها في علاج ال cardiogenic we septic shock we el hypovalemic la2 :S
ويعني ايه septic aslan ?
سؤال تانى بقي في البلوكير بتاعة ال beta
ازاي بتعالج ال hypertension
؟
وازاي ال selctive beta 1 blocker بنستخدمها في علاج ال heart failure ؟
Mohamed Sakr
alpha 1 agonist contra'indicated in cardiogenic shock due to present of decreasing blood flow to organs and alpha 1 agonist make vasoconstriction decreasing blood flow to organs .
septic shock is presence of infection in the bovy making release of inflammatory madiators making severe vasodilation making hypotension must give alpha 1 agonist to make vasoconstriction and increasing blood pressure
dopamine is first choice in treatment of cardiogenic shock due to making increasing cardiac output through acting on beta 1 receptor and making vasodilation of blood vessels making increase of blood flow through acting on dopamine receptor
betablocker treatment hypertension due to blocking beta 1 receptor in the heart making decreasing in heart rate + decreasing cardiac output and blocking of bata 1 receptor in the kidney juxtaglomerular cells decreasing renin and decreasing angiotensin II make V.D + blocking of beta 2 receptor in the sympathetic nerve decreasing NA release + inhibit sympathetic outflow centrally from sympathetic center
use carvidelol , metoprolol , bisoprolol in treatment of heart failure due to prevention of excessive sympathetic discharge on the heart + carvidelol making alpha blocker activity making V.D in artery and veins making decreasing afterload and preload making resting of heart47 minutes ago · - Talta Fopcu مهمة جداااااااا والدكتور محمد صقر مجمعها بطريقة ممتازة
* M1 , M3 , Alpha 1 is Gq increasing DAG+IP3 Increasing Ca+2 bind to calmodulin .
* Beta 1 , Beta 2 , Beta 3 is Gs activate adenyl cyclase increasing CAmp .
* M2 , Alpha 2 is Gi which inhibit adenyl cyclase decreasing CAmp .46 minutes ago · · 2 - Talta Fopcu * DAG is lipophilic present in lipid membrane making activation of phospholipase C .
* IP3 is hydrophilic present in the cytoplasm making increasing of Ca+2 .44 minutes ago · · 1
Mai Hashad Nn: nicotinic neural & Nm: Nicotinic muscle & D: dopamine & CGMP:cyclic guanosine monophosphate & EDRF:endothelium derived releasing factor
42 minutes ago ·
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